Close
  Indian J Med Microbiol
 

Figure 1: Cellular mechanism of ischemic preconditioning. Adenosine A1 and A3 (A1/A3), bradykinin2 (B2), δ1-opioid (δ1), α1-adrenergic (α1), β-adrenergic (β) receptors, phospholipase C (PLC), guanine nucleotide-binding proteins (Gi protein), diacylglycerol (DAG), inositol trisphosphate (IP3), protein kinase C (PKC), ATP-sensitive K+ channels (KATP channels), mitogen-activated protein kinases (MAPK). NO, nitric oxide, NOS, nitric oxide synthase, PIP2, phosphatidylinositol bisphosphate, ROS, reactive oxygen species, SR, sarcoplasmic reticulum 53.

Figure 1: Cellular mechanism of ischemic preconditioning. Adenosine A1 and A3 (A1/A3), bradykinin2 (B2), δ1-opioid (δ1), α1-adrenergic (α1), β-adrenergic (β) receptors, phospholipase C (PLC), guanine nucleotide-binding proteins (Gi protein), diacylglycerol (DAG), inositol trisphosphate (IP3), protein kinase C (PKC), ATP-sensitive K+ channels (K<sub>ATP<sub> channels), mitogen-activated protein kinases (MAPK). NO, nitric oxide, NOS, nitric oxide synthase, PIP2, phosphatidylinositol bisphosphate, ROS, reactive oxygen species, SR, sarcoplasmic reticulum 53.